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Natasha Jaiswal

  • Assistant Professor, Department of Health and Kinesiology

Office and Contact

Room: LAMB 306c

Email: jaiswa19@purdue.edu

Phone: 765-496-0569


Education
PhD Central Drug Research Institute, India Biochemistry 2015
 
Research Interests
Skeletal muscle and NMJ plasticity in physiological and pathological conditions, glucose homeostasis, body composition, obesity and sarcopenia
 
Teaching Interests
Physiology, metabolism and biochemistry
 
Selected Grants
2025 - Clifford B. Kinley Trust Award, Purdue University
2024 - Faculty Research Development grant, Centre on Aging and the Life Course, Purdue University
2023 - NIA P30 mentorship award, Oklahoma Nathan Shock Center
2021 - Pilot Career Development Grant, San Antonio Nathan Shock Center
 
Selected Publications
N Jaiswal, N. R. Lin, C. F. Lehnen, A Tariq, L. L. Lukov and C Zhang. AKT Signaling Regulates
Agrin-Mediated Acetylcholine Receptor Surface Density. Medicina 62(3), 456 (2026).

N. Jaiswal, M. Gavin, L. Lantier, O. Y. Y. Ong, D. H. Wasserman, P. M. Titchenell. Divergent AKT Signaling
Mechanisms Regulate GLUT4 Translocation and Glucose Uptake in Skeletal-Muscle and Adipose Tissue.
Journal of Cachexia, Sarcopenia and Muscle (In communication, final round).
 
W. D. Lee, D. R. Weilandt, L. Liang, M. R. MacArthur, N. Jaiswal, C. G. Mann, Q. C. Craig J. Hunter, R-P
Ryseck, W. Lu, A. M. Oschmann, A. J. Cowan, T. A. TeSlaa, C. R. Bartman, C. Jang, J. A. Baur, P. M.
Titchenell, and J. D. Rabinowitz. Lactate-glucose-fat regulatory circuitry mediating lactate homeostasis.
Cell Metabolism (2024).

N Jaiswal, M Gavin, E Loro, J Sostre-Colón, PA Roberson, K Uehara, N R Fuentes, M Neinast, Z Arany,
S R Kimball, T S Khurana, P M Titchenell. AKT controls protein synthesis and oxidative metabolism via
combined mTORC1 and FOXO1 signaling to govern muscle physiology. Journal of Cachexia, Sarcopenia
and Muscle (2021).
 
E Nunn, N Jaiswal, M Gavin, K Uehara, M Stefkovich, K Drareni, R Calhoun, M Lee, C Holman, J Baur,
S Patrick, P. Titchenell. Antibody Blockade of Activin Type II Receptors Preserves Skeletal Muscle Mass
and Enhances Fat Loss During GLP-1 Receptor Agonism. Molecular metabolism (Accepted, January,
2024).